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) Touch me, tease me, kiss me, please me I give it to you just how you like it, girl You're now I'm rockin' with the best Tre pound on my hip, teflon on my chest They say I'm no good, cause I'm so hood Rich folks do not want me around Cause shit might pop off, and if shit pop off Somebody gon' get laid the fuck out They call me new money, say I have no class I'm from the bottom, I came up too fast The hell if I care, I'm just here to get my cash Bougie-ass bitches, you can kiss my ass [Hook: 50 Cent] This, is, how, we, do We make a move and act a fool while we up in the club This, is, how, we, do Nobody do it like we do it so show us some love This, is, how, we, do We make a move and act a fool while we up in the club This, is, how, we, do Nobody do it like we do it so show us some love [Verse 3: The Game] I put gold Daytonas on that cherry six-fo' White walls so clean it's like I'm ridin' on Vogues Hit one switch mayne, that ass so low Cali got niggas in New York ridin' on hundred spokes Touch me, tease me, kiss me, please me I give it to you just how you like it, girl You're now I'm rockin' with the best Four pound on my hip, gold chain on my chest (ahh!

) [Verse 4: 50 Cent & The Game] 50, uhh, Bentley, uhh Em came and got a nigga fresh out the slum Automatic, gun, fuck 'em one-on-one We wrap up your punk ass, stunt and you done Homie, it's Game time (you ready?

The acute effects of ketamine cause cognitive impairment, including reductions in vigilance, verbal fluency, short-term memory, and executive function, as well as schizophrenia-like perceptual changes.

Urinary tract symptoms have been collectively referred as "ketamine-induced ulcerative cystitis" or "ketamine-induced vesicopathy", and they include urge incontinence, decreased bladder compliance, decreased bladder volume, detrusor overactivity, and painful blood in urine.

This suggests the infrequent use of ketamine does not cause cognitive deficits, and that any deficits that might occur may be reversible when ketamine use is discontinued.

However, abstinent, frequent, and infrequent users all scored higher than controls on a test of delusional symptoms.Bilateral hydronephrosis and renal papillary necrosis have also been reported in some cases.The pathogenesis of papillary necrosis has been investigated in mice, and mononuclear inflammatory infiltration in the renal papilla resulting from ketamine dependence has been suggested as a possible mechanism.The first large-scale, longitudinal study of ketamine users found current frequent (averaging 20 days/month) ketamine users had increased depression and impaired memory by several measures, including verbal, short-term memory, and visual memory.Current infrequent (averaging 3.25 days/month) ketamine users and former ketamine users were not found to differ from controls in memory, attention, and psychological well-being tests.Currently, ketamine is not approved for the treatment of depression, and so this is an off-label use.As of June 2017, esketamine, the S( ) enantiomer of ketamine, is in phase III clinical trials for intranasal treatment of depression.Ketamine is an analgesic that is most effective when used alongside a low-dose opioid; because, while it does have analgesic effects by itself, the doses required for adequate pain relief when it is used as the sole analgesic agent are considerably higher and far more likely to produce disorienting side effects.Meta-analyses have shown overwhelming clinical evidence to support the acute efficacy of ketamine in severely unwell populations, but lack consensus on optimal dosing and the effect of long-term treatment.One such study administered daily ketamine doses consistent with typical recreational doses (1 mg/kg IV) to adolescent cynomolgus monkeys for varying periods of time.Decreased locomotor activity and indicators of increased cell death in the prefrontal cortex were detected in monkeys given daily injections for six months, but not those given daily injections for one month.

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